Previous research shows risk for ADHD can increase in the first year following severe traumatic brain injury in kids, but what are risks over the long term and after mild to moderate injury?

Traumatic brain injury is a leading cause of death and disability in the world. Blunt force trauma to the brain, often from a bad fall or traffic accident, accounts for the deaths of over 61,000 ...

Targeted oxidation-blocker prevents secondary damage after traumatic brain injury, study suggests. ScienceDaily. Retrieved June 2, 2025 from www.sciencedaily.com / releases / 2012 / 08 ...

Traumatic brain injuries (TBI) contribute to many deaths each year, and can lead to the development of secondary mental health problems. The Centre for Disease Control has reported that ...

Traumatic brain injury (TBI) doesn’t stop at impact. TBI is a spectrum, from mild to severe, with different symptoms and outcomes. ... Secondary injuries occur as the brain tries to recover.

According to a research team, treatment with a specific agent prevented the secondary damage of severe traumatic brain injury and preserved function that would otherwise have been impaired. About ...

A recently published study sheds light on how spreading depolarization can produce secondary damage after traumatic brain injury (TBI), and a second study presents a technique for the noninvasive ...

Not only do patients with traumatic brain injury (TBI) require the usual health management services of community-based primary care providers ... This website is an auxiliary resource for PCPs and ...

Researchers in the University of Cincinnati Department of Neurosurgery are working to find a way to stop the wave of secondary injury that follows an initial blow to the head.

Doctors classify Traumatic Brain Injury (TBI) into three categories based on the severity of the injury. ... The initial focus is to stabilize the injured person to minimize secondary complications.

Risk factors in psychosis secondary to traumatic brain injury. The Journal of neuropsychiatry and clinical neurosciences, 13(1), 61-69. Fujii, D. (2002).